Purpose: Peri-implantitis is defined by an inflammation that occurs at the soft tissue and the alveolar bone around the site of an implant.
The failure of implant placement or the loss of the supporting bone may occur due to peri-implantitis. From a pathological perspective,
A. actinomycetemcomitans, Capnocytophaga sp., F. nucleatum, or other infectious agent is seen as the cause of the peri-implantitis.
To remove the cause of the disease, many non-invasive methods are being studied. In this study, we demonstrated decontamination
of the implant surface and its surroundings through experiments using diode laser products in existing products and diode laser experiments
that studied the effect on the decreasing peri-implants.
Materials and Methods: 12-week-old male Sprague-Dawley rats weighing 300 g were purchased. The size of 1.2 * 4 mm hole was
drilled into the hard plate of the maxillary bone to insert titanium screw implant. Test groups were divided into control, titanium screw
implant group, peri-implantitis group, and laser-treated peri-implantitis group. Infection levels around the site of implants were checked
with SEM. The degree of microbial reproduction was checked through real-time PCR (qPCR) for each group.
Results: The use of 808 nm Diode laser (0.5 w, 15 seconds in continuous mode) to treat the inflammation caused by peri-implantitis
in the soft tissue and the alveolar bone resulted in an effective reduction in the number of bacteria without the surface denaturation of
the implant due to the laser. In comparison to the existing products, such as Picaso Diode laser (810 nm) and Bison Diode laser (808
nm), both products have shown effectiveness in eliminating bacteria. In particular, 808 nm Diode laser showed equal effectiveness to
the 810 nm Diode laser at a lower temperature.
Conclusion: Peri-implantitis was treated in the inflammation region of soft tissue and alveolar bone using the 808 nm and 810 nm Diode
laser in continuous mode at 0.5 w for 15 seconds. As a result, laser-induced excessive heat generation or denaturation on the implant
surfaces did not occur and the number of bacteriadecreased. (JOURNAL OF DENTAL IMPLANT RESEARCH 2016;35(1):1-8)