- 근육 구조특성과 선행 신장성 수축에 의한 항정상태 등척성 근력 증대와의 연관성에 대한 연구
- ㆍ 저자명
- 이해동,이중현,Lee. Hae-Dong,Lee. Jung-Hyun
- ㆍ 간행물명
- 한국운동역학회지
- ㆍ 권/호정보
- 2007년|17권 3호|pp.61-68 (8 pages)
- ㆍ 발행정보
- 한국운동역학회
- ㆍ 파일정보
- 정기간행물| PDF텍스트
- ㆍ 주제분야
- 기타
When an active muscle is stretched, its steady-state isometric force following stretch is greater than that of a purely isometric contraction as the corresponding muscle length, referred to as force enhancement (FE). The purpose of this study was to investigate possible effects of muscle architecture on the FE. While subject performed maximal isometric dorsiflexion (REF) and isometric-stretch-isometric dorsiflexion (ECC) contractions, ankle joint angle and dorsiflexion torque using a dynamometer and electromyography of the tibialis anterior and the medical gastrocnemius muscles were measure. Simultaneously, real-time ultrasound images of the tibialis anterior were acquired. Regardless of the speed of stretch of the ECC contractions. the torques produced during the isometric phase following stretch ($37.3{pm}1.5;Nm$ ($10{pm}3%$ FE) and $38.3{pm}1.5$ ($12{pm}3%$ FE) for the ECC contractions with $15^{circ}$/s and $45^{circ}$/s stretch speeds, respectively) were greater than those of the REF contractions ($34.5{pm}2.5;Nm$). Moreover, the amount of FE was found to be stretch speed dependent. Angles of pennation ($alpha$) during the isometric phase following stretch were the same for the REF ($15{pm}1^{circ}$) and the ECC ($14{pm}1^{circ}$(LS), $15{pm}1^{circ}$(LF)). During the same phase, muscle thicknesses were the same ($14.9{pm}0.6$, and $14.9{pm}0.5;mm$ for the REF and the ECC contractions, respectively). For a large limb muscle, the tibialis anterior muscle, a similar amount of force enhancement was observed as did for other human skeletal muscles. Architectural variables, pennation angle and thickness, were not systematically different between the REF and ECC contractions when FE occurred. Therefore, the results of this study suggest that muscle architecture may have little influence on the production of FE.