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A New Triterpenoid from Panax ginseng Exhibits Cytotoxicity through p53 and the Caspase Signaling Pathway in the HepG2 Cell Line
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  • A New Triterpenoid from Panax ginseng Exhibits Cytotoxicity through p53 and the Caspase Signaling Pathway in the HepG2 Cell Line
  • A New Triterpenoid from Panax ginseng Exhibits Cytotoxicity through p53 and the Caspase Signaling Pathway in the HepG2 Cell Line
저자명
Huang. Jian,Tang. Xiao-Hui,Ikejima. Takashi,Sun. Xiu-Jia,Wang. Xiao-Bo,Xi. Rong-Gang,Wu. Li-Jun
간행물명
Archives of pharmacal research : a publication of the Pharmaceutical Society of Korea
권/호정보
2008년|31권 3호|pp.323-329 (7 pages)
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대한약학회
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정기간행물|ENG|
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이 논문은 한국과학기술정보연구원과 논문 연계를 통해 무료로 제공되는 원문입니다.
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기타언어초록

A new triterpenoid, 20(R),22$({zeta})$,24(S)-dammar-25(26)-ene-$3{eta},6{alpha},12{eta}$,20,22,24-hexanol (1), and three known triterpenoids, ${eta}$-D-glucopyranoside,($3{eta},12{eta}$)-12,20-dihydroxydammar-24-en-3-yl,6-acetate (2), 20(R)-ginsenoside $Rg_3$ (3), and 20(R)-ginsenoside $Rg_2$ (4), were isolated from the leaves of Panax ginseng. Their structures were determined by chemical analysis and spectral methods (IR, 1D and 2D NMR, HR-ESI-MS). Compounds 1-4 were exhibited various degrees of cytotoxicity in the human hepatoma cell line, HepG2. Compound 1 had the highest cytotoxic Potency, with an $IC_{50}$ value of 20.1 ${mu}M$, by stimulating p53-mediated cell cycle arrest at the G1 to S phase transition, leading to apoptosis via activation of the caspase signaling pathway.