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Sulfasalazine Induces Autophagic Cell Death in Oral Cancer Cells via Akt and ERK Pathways
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  • Sulfasalazine Induces Autophagic Cell Death in Oral Cancer Cells via Akt and ERK Pathways
  • Sulfasalazine Induces Autophagic Cell Death in Oral Cancer Cells via Akt and ERK Pathways
저자명
Han. Hye-Yeon,Kim. Hyungwoo,Jeong. Sung-Hee,Lim. Do-Seon,Ryu. Mi Heon
간행물명
Asian Pacific journal of cancer prevention : APJCP
권/호정보
2014년|15권 16호|pp.6939-6944 (6 pages)
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아시아태평양암예방학회
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정기간행물|ENG|
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이 논문은 한국과학기술정보연구원과 논문 연계를 통해 무료로 제공되는 원문입니다.
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기타언어초록

Sulfasalazine (SSZ) is an anti-inflammatory drug that has been used to treat inflammatory bowel disease and rheumatoid arthritis for decades. Recently, some reports have suggested that SSZ also has anti-cancer properties against human tumors. However, little is known about the effects of SSZ on oral cancer. The aim of this study was to investigate the anti-cancer effects of SSZ in oral squamous cell carcinoma (OSCC) cells and to elucidate the mechanisms involved. The authors investigated the anti-proliferative effect of SSZ using the MTT method in HSC-4 cells (an OSCC cell line). Cell cycle analysis, acidic vesicular organelle (AVO) staining, monodansylcadaverine (MDC) staining and Western blotting were also conducted to investigate the cytotoxic mechanism of SSZ. SSZ significantly inhibited the proliferation of HSC-4 cells in a dose-dependent manner. In addition, SSZ induced autophagic cell death, increased microtubule-associated protein 1 light chain (MAP1-LC; also known as LC) 3-II levels, as well as induced punctate AVO and MDC staining, resulted in autophagic cell death. Furthermore, these observations were accompanied by the inhibition of the Akt pathway and the activation of ERK pathway. These results suggest that SSZ promotes autophagic cell death via Akt and ERK pathways and has chemotherapeutic potential for the treatment of oral cancer.