황화녹두의 하배축에서 LiCl는에틸렌 생성을 유도하였다. 리튬이온은 또 IAA나 ACC에 의하여 유도된 에틸렌 생성도 증가시켰다. 리튬이온에 의한 IAA유도에틸렌 생성의 증가는 IAA 흡입에 대한 영향의 결과는 아니라는 결론을 얻었다. IAA의 높은 농도(1 mM)로 생성되는 에틸렌의 생성은 리튬이온에 의하여 오히려 억제되는 양상을 보였다. $^3H$-uridine과 $^3H$-leucine을 이용한 실험으로 리튬이온은 이 조직에서 RNA 및 단백질 합성을 억제시킨다는 결과를 얻였다. 이러한 실험결과들을 토대로 리튬이온이 에틸렌 생합성 경로에서 어디에 작용 하는지를 고찰하였다.
Non-osmotic concentrations of LiCl promoted ethylene production from hypocotyl segments of etiolated mung bean seedlings. Indole-3-acetic acid (IAA)-induced, as well as 1-aminocyclopropane-1-carboxylic acid (ACC)-based ethylene production was further promoted by treatment of the segments with lithium ions. Results of experiments using labelled IAA indicated that the promotive effect of $Li^+$ on auxin-induced ethylene production was not a result of possible mediation of auxin uptake in to the cell by the cation. The lithium effect on auxin-induced ethylene production was found to be reversed at high concentrations (eg. 1 mM) of added IAA. Syntheses of both protein and RNA in the tissue as measured by in vivo incorporations of $^3H$-leucine and $^3H$-uridine respectively into TCA precipitates were likewise inhibited by lithium ions. Possible sites of lithium action in the pathway of ethylene biosynthesis will be discussed.
